– credit Tony Kershaw via SWNS
Tipah and her litter of cubs – credit Tony Kershaw via SWNS


– credit Tony Kershaw via SWNS
Tipah and her litter of cubs – credit Tony Kershaw via SWNS


Carole Grieg testing the CrossSense AI glasses – SWNS
Warning appears on the lenses of the CrossSense AI glasses (GNN screenshot of SWNS/CrossSense video)
Screenshot of Wispy AI in the midst of interacting with user of theCrossSense AI glasses, discussing care of a houseplant (Still from SWNS video)

Masaya at the Liverpool Vet. Hospital where she underwent surgery – credit, Chester Zoo via SWNS
What can you see right now? This might seem like a silly question, but what enters your consciousness is not the whole story when it comes to vision. A great deal of visual processing in the brain goes on well below our conscious awareness.
Some studies have probed the unconscious depths of vision. One source of evidence comes from the neurological condition known as blindsight, which is caused by damage to areas of the brain involved in processing visual information. People with blindsight report that they are unable to see, either entirely or in a portion of their visual field. However, when asked to guess what is there, they can often do so with remarkable accuracy.
For example, in an experiment published in 2004 on someone with blindsight, a black bar was displayed in the portion of the visual field to which the person was blind. The person was asked to “guess” whether the bar was vertical or horizontal.
Despite denying any conscious awareness of the bar, the participant could answer correctly at a level well above chance. The participant even showed evidence of being able to pay attention to the bar – they were faster to respond when an arrow (placed in a healthy area of their visual field) correctly indicated the location of the bar.
The most popular interpretation (though not the only one) is that people with blindsight can see these objects, but not see them consciously. They see what is there, but it all goes on unconsciously, below their awareness.
The phenomenon of inattentional blindness seems to show you can see without the information crossing into your consciousness. Anyone can experience inattentional blindness. The phenomenon has been known about for a long time, but we can most easily get a handle on it by looking at a well-known experiment reported in 1999.
In this experiment, participants are shown a video of people playing basketball, and told to count the number of passes between the players wearing a white shirt. If you’ve never done this before, I urge to you stop reading now and watch the video.
In many cases, people are so busy counting the passes that they completely miss a large gorilla walking across the middle of the scene and beating its chest, then walking off. The gorilla’s right there, in the centre of your visual field. Light from the gorilla enters your eyes, and is processed in the visual system, but somehow you missed it, because you weren’t paying attention to it.
The gorilla has more to teach us. In another experiment reported in 2013, radiologists were given a series of lung scans. They were told to look for nodules (which show up as small light coloured circles) on each scan. In one of the scans, a large picture of a dancing gorilla was superimposed on top of the lung scan. In this study, 83% of the radiologists failed to spot it, even though it was 48 times bigger than the average nodule they were looking for. Some of them even looked directly at the gorilla and still didn’t notice it!
The interpretation of these experiments is controversial. Some scientists suggest that in these kinds of cases, you consciously see the gorilla, but immediately forget it (although a dancing gorilla in someone’s lung doesn’t seem like the kind of thing you’d forget). Others argue that you see the gorilla, but the information never made its way into consciousness. You saw the gorilla, but unconsciously.
Let’s assume that in the case of blindsight, and inattentional blindness, the information is seen, but didn’t make it all the way to consciousness. Then, the question is: what makes some information conscious, rather than the information that stays unconscious? This is one of the central questions for consciousness studies in philosophy, psychology and neuroscience.
There’s no agreement on which is the best theory of consciousness, but in my opinion, the strongest contender is the global neuronal workspace theory.
According to this theory, consciousness is all to do with a particular area of the brain which is the seat of the “workspace”. The workspace is a system with a small capacity, so it can’t hold a lot of information at any one time. The job of the workspace is to take unconscious information and broadcast it to lots of different networks all across the brain. Global neuronal workspace theorists say that broadcasting the information in this way is what makes it conscious.
The job of the workspace is to act like the brain’s loudspeaker, and consciousness is the information that gets broadcast. The workspace takes unconscious information and boosts it so that many of the different systems in the brain hear about it and can use that information in their own processes. The late philosopher Daniel Dennett used to call consciousness “fame in the brain”. The workspace idea is similar.
One of the most striking implications of the global neuronal workspace theory is how little information makes it to consciousness. Since the workspace has quite a small capacity, it follows that we can only ever be conscious of a little at a time. We might think there’s a rich visual world in front of us, full of details, all of which we’re conscious of, but really – according to the theory – we’re only ever conscious of a small portion of that.
Some philosophers and scientists have objected to the theory on these grounds. They suggest that consciousness “overflows” the workspace: we are conscious of more information than can “fit” into the workspace at any one time. Even with these debates still ongoing, I think the global neuronal workspace theory gives us a reasonably clear answer to the question of what consciousness is for, and how it interacts with other systems in the brain.
In our brains, consciousness is only the tip of a very large iceberg. But the global neuronal workspace theory might give us insight into what makes that tip so special.![]()
Henry Taylor, Associate Professor, Department of Philosophy, University of Birmingham
This article is republished from The Conversation under a Creative Commons license. Read the original article.

Flexible inkjet printed E-textile – Credit: Marzia Dulal
Gloves with e-textile sensors monitoring heart rate – Credit: Marzia Dulal
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But in our new study my colleagues and I found that the changing climate was driving changes in the polar bear genome, potentially allowing them to more readily adapt to warmer habitats. Provided these polar bears can source enough food and breeding partners, this suggests they may potentially survive these new challenging climates.
We discovered a strong link between rising temperatures in south-east Greenland and changes in polar bear DNA. DNA is the instruction book inside every cell, guiding how an organism grows and develops. In processes called transcription and translation, DNA is copied to generate RNA (molecules that reflect gene activity) and can lead to the production of proteins, and copies of transposons (TEs), also known as “jumping genes”, which are mobile pieces of the genome that can move around and influence how other genes work.
In carrying out our recent research we found that there were big differences in the temperatures observed in the north-east, compared with the south-east regions of Greenland. Our team used publicly available polar bear genetic data from a research group at the University of Washington, US, to support our study. This dataset was generated from blood samples collected from polar bears in both northern and south-eastern Greenland.
Our work built on the Washington University study which discovered that this south-eastern population of Greenland polar bears was genetically different to the north-eastern population. South-east bears had migrated from the north and became isolated and separate approximately 200 years ago, it found.
Researchers from Washington had extracted RNA from polar bear blood samples and sequenced it. We used this RNA sequencing to look at RNA expression — the molecules that act like messengers, showing which genes are active, in relation to the climate. This gave us a detailed picture of gene activity, including the behaviour of TEs. Temperatures in Greenland have been closely monitored and recorded by the Danish Meteorological Institute. So we linked this climate data with the RNA data to explore how environmental changes may be influencing polar bear biology.
From our analysis we found that temperatures in the north-east of Greenland were colder and less variable, while south-east temperatures fluctuated and were significantly warmer. The figure below shows our data as well as how temperature varies across Greenland, with warmer and more volatile conditions in the south-east. This creates many challenges and changes to the habitats for the polar bears living in these regions.
In the south-east of Greenland, the ice-sheet margin, which is the edge of the ice sheet and spans 80% of Greenland, is rapidly receding, causing vast ice and habitat loss.
The loss of ice is a substantial problem for the polar bears, as this reduces the availability of hunting platforms to catch seals, leading to isolation and food scarcity. The north-east of Greenland is a vast, flat Arctic tundra, while south-east Greenland is covered by forest tundra (the transitional zone between coniferous forest and Arctic tundra). The south-east climate has high levels of rain, wind, and steep coastal mountains.
Temperature across Greenland and bear locations
Author data visualisation using temperature data from the Danish Meteorological Institute. Locations of bears in south-east (red icons) and north-east (blue icons). CC BY-NC-NDOver time the DNA sequence can slowly change and evolve, but environmental stress, such as warmer climate, can accelerate this process.
TEs are like puzzle pieces that can rearrange themselves, sometimes helping animals adapt to new environments. In the polar bear genome approximately 38.1% of the genome is made up of TEs. TEs come in many different families and have slightly different behaviours, but in essence they all are mobile fragments that can reinsert randomly anywhere in the genome.
In the human genome, 45% is comprised of TEs and in plants it can be over 70%. There are small protective molecules called piwi-interacting RNAs (piRNAs) that can silence the activity of TEs.
Despite this, when an environmental stress is too strong, these protective piRNAs cannot keep up with the invasive actions of TEs. In our work we found that the warmer south-east climate led to a mass mobilisation from these TEs across the polar bear genome, changing its sequence. We also found that these TE sequences appeared younger and more abundant in the south-east bears, with over 1,500 of them “upregulated”, which suggests recent genetic changes that may help bears adapt to rising temperatures.
Some of these elements overlap with genes linked to stress responses and metabolism, hinting at a possible role in coping with climate change. By studying these jumping genes, we uncovered how the polar bear genome adapts and responds, in the shorter term, to environmental stress and warmer climates.
Our research found that some genes linked to heat-stress, ageing and metabolism are behaving differently in the south-east population of polar bears. This suggests they might be adjusting to their warmer conditions. Additionally, we found active jumping genes in parts of the genome that are involved in areas tied to fat processing – important when food is scarce. This could mean that polar bears in the south-east are slowly adapting to eating the rougher plant-based diets that can be found in the warmer regions. Northern populations of bears eat mainly fatty seals.
Overall, climate change is reshaping polar bear habitats, leading to genetic changes, with south-eastern bears evolving to survive these new terrains and diets. Future research could include other polar bear populations living in challenging climates. Understanding these genetic changes help researchers see how polar bears might survive in a warming world – and which populations are most at risk.
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Alice Godden, Senior Research Associate, School of Biological Sciences, University of East Anglia
This article is republished from The Conversation under a Creative Commons license. Read the original article.


Courtesy of Oliver Chu family

credit – Adam Kay, Twycross Zoo / SWNSWe spend nearly a third of our lives asleep, yet sleep is anything but wasted time. Far from being passive downtime, it is an active and essential process that helps restore the body and protect the brain. When sleep is disrupted, the brain feels the consequences – sometimes in subtle ways that accumulate over years.
In a new study, my colleagues and I examined sleep behaviour and detailed brain MRI scan data in more than 27,000 UK adults between the ages of 40 and 70. We found that people with poor sleep had brains that appeared significantly older than expected based on their actual age.
What does it mean for the brain to “look older”? While we all grow chronologically older at the same pace, some people’s biological clocks can tick faster or slower than others. New advances in brain imaging and artificial intelligence allow researchers to estimate a person’s brain age based on patterns in brain MRI scans, such as loss of brain tissue, thinning of the cortex and damage to blood vessels.
In our study, brain age was estimated using over 1,000 different imaging markers from MRI scans. We first trained a machine learning model on the scans of the healthiest participants – people with no major diseases, whose brains should closely match their chronological age. Once the model “learned” what normal ageing looks like, we applied it to the full study population.
Having a brain age higher than your actual age can be a signal of departure from healthy ageing. Previous research has linked an older-appearing brain to faster cognitive decline, greater dementia risk and even higher risk of early death.
Sleep is complex, and no single measure can tell the whole story of a person’s sleep health. Our study, therefore, focused on five aspects of sleep self-reported by the study participants: their chronotype (“morning” or “evening” person), how many hours they typically sleep (seven to eight hours is considered optimal), whether they experience insomnia, whether they snore and whether they feel excessively sleepy during the day.
These characteristics can interact in synergistic ways. For example, someone with frequent insomnia may also feel more daytime sleepiness, and having a late chronotype may lead to shorter sleep duration. By integrating all five characteristics into a “healthy sleep score”, we captured a fuller picture of overall sleep health.
People with four or five healthy traits had a “healthy” sleep profile, while those with two to three had an “intermediate” profile, and those with zero or one had a “poor” profile.
When we compared brain age across different sleep profiles, the differences were clear. The gap between brain age and chronological age widened by about six months for every one point decrease in healthy sleep score. On average, people with a poor sleep profile had brains that appeared nearly one year older than expected based on their chronological age, while those with a healthy sleep profile showed no such gap.
We also considered the five sleep characteristics individually: late chronotype and abnormal sleep duration stood out as the biggest contributors to faster brain ageing.
A year may not sound like much, but in terms of brain health, it matters. Even small accelerations in brain ageing can compound over time, potentially increasing the risk of cognitive impairment, dementia and other neurological conditions.
The good news is that sleep habits are modifiable. While not all sleep problems are easily fixed, simple strategies: keeping a regular sleep schedule; limiting caffeine, alcohol and screen use before bedtime; and creating a dark and quiet sleep environment can improve sleep health and may protect brain health.
One explanation may be inflammation. Increasing evidence suggests that sleep disturbances raise the level of inflammation in the body. In turn, inflammation can harm the brain in several ways: damaging blood vessels, triggering the buildup of toxic proteins and speeding up brain cell death.
We were able to investigate the role of inflammation thanks to blood samples collected from participants at the beginning of the study. These samples contain a wealth of information about different inflammatory biomarkers circulating in the body. When we factored this into our analysis, we found that inflammation levels accounted for about 10% of the connection between sleep and brain ageing.
Another explanation centres on the glymphatic system – the brain’s built-in waste clearance network, which is mainly active during sleep. When sleep is disrupted or insufficient, this system may not function properly, allowing harmful substances to build up in the brain.
Yet another possibility is that poor sleep increases the risk of other health conditions that are themselves damaging for brain health, including type 2 diabetes, obesity and cardiovascular disease.
Our study is one of the largest and most comprehensive of its kind, benefiting from a very large study population, a multidimensional measure of sleep health, and a detailed estimation of brain age through thousands of brain MRI features. Though previous research connected poor sleep to cognitive decline and dementia, our study further demonstrated that poor sleep is tied to a measurably older-looking brain, and inflammation might explain this link.
Brain ageing cannot be avoided, but our behaviour and lifestyle choices can shape how it unfolds. The implications of our research are clear: to keep the brain healthier for longer, it is important to make sleep a priority.![]()
Abigail Dove, Postdoctoral Researcher, Neuroepidemiology, Karolinska Institutet
This article is republished from The Conversation under a Creative Commons license. Read the original article.